How cognitive support in the office lowers the risk of dementia in old age

Brain activities that stimulate cognitive support, such as reading, writing and puzzling, could delay the outbreak of Alzheimer's disease and dementia by 5 years. This emerges from a recently published study that is the most commonCauses of dementiadealt. The new results of the scientific work also appear in the magazine Neurology. In the current report, the study authors explain the connection between intellectual stimulation and dementia.

Prevent neurodegenerative diseases by cognitive funding

According to the study authors, people with mentally stimulating jobs have a lower risk of dementia in later life. Specifically, the new research work connected the advantages of cognitive activities in the workplace with a reduced risk of dementia. More precisely, this is a meta -analysis that examined existing data of more than 123,000 people for 17 years. The research team focused on the relationship between factors associated with work and chronic diseases, disability and mortality. Finally, the results showed that people who had to solve more issues at work had a lower risk of dementia. The study results also remained convincing, taking into account the risk factors for dementia. However, the researchers also pointed out that this relationship neither varies between men and women nor between people below and over 60 years. However, it seems that the connection between Alzheimer's disease is more pronounced than in other forms of dementia.

Cognitive funding was also associated with lower concentrations of three proteins that are connected to dementia in adulthood. According to the study authors, this provides possible references to underlying biological mechanisms. They assume that mental stimulation maintains a low content of certain proteins. Accordingly, these prevent brain cells preventing new connections between the neurons. The knowledgethis studythat cognitive stimulation is connected to lower mirrors of these plasma proteins, which may inhibit the so -called axonogenesis and synaptogenesis and increase the risk of dementia, could provide information on the underlying biological mechanisms.